EP06
Dietary Salt Intake and Risk of Pediatric MS: A Prospective Case-Control Study

Thursday, May 29, 2014
Trinity Exhibit Hall
Jamie McDonald, MS , UCSF Pediatric MS Center, San Francisco, CA
Jennifer Graves, MD, PhD , UCSF Pediatric MS Center, San Francisco, CA
Sabeen Lulu, MD , UCSF Pediatric MS Center, San Francisco, CA
Amy Waldman, MD, MSCE , Department of Neurology, University of Pennsylvania, Philadelphia, PA
Anita Belman, MD , Department of Neurology, SUNY Stony Brook, Stony Brook, NY
Benjamin Greenberg, MD, MHS , Department of Neurology, UT Southwestern, Dallas, TX
Bianca Weinstock-Guttman, MD , Jacobs MS Center, Buffalo, NY
Greg Aaen, MD , Department of Child Neurology, Loma Linda University, Loma Linda, CA
Jan Mendelt-Tillema, MD , Department of Neurology, Mayo Clinic, Rochester, MN
Janace Hart, BA , UCSF Pediatric MS Center, San Francisco, CA
Jayne Ness, MD , Alabama Pediatric MS Center, Birmingham, AL
Jennifer Rubin, MD , Department of Pediatric Neurology, Northwestern Feinberg School of Medicine, Chicago, IL
Lauren Krupp, MD , Department of Neurology, SUNY Stony Brook, Stony Brook, NY
Mark Gorman, MD , Massachusetts General Hospital, Partners Pediatric MS Center, Boston, MA
Moses Rodriguez, MD , Department of Neurology, Mayo Clinic, Rochester, MN
Tanuja Chitnis, MD , Massachusetts General Hospital, Partners Pediatric MS Center, Boston, MA
Timothy Simmons, MStat , Department of Pediatrics, University of Utah, Salt Lake City, UT
T. Charles Casper, PhD , Department of Pediatrics, University of Utah, Salt Lake City, UT
John Rose, MD , Department of Neurology, University of Utah, Salt Lake City, UT
Emmanuelle Waubant, MD, PhD , UCSF Pediatric MS Center, San Francisco, CA
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Background:

Environmental and dietary factors have become increasingly recognized in the past decades as potential risk factors for developing multiple sclerosis (MS). Pediatric MS offers a unique opportunity to study such factors, due to temporal proximity at the time of diagnosis to the exposure, thereby minimizing recall bias. High salt intake has been shown to increase disease onset and progression in recent animal studies. Whether these results are applicable to human disease is currently unknown. 

Objectives:

To determine if dietary salt intake is higher in a multi-center cohort of pediatric MS subjects compared to pediatric controls.

Methods:

A prospective case-control study was performed with pediatric-onset MS patients (first clinical attack before 18 years of age) who were seen within 2 years of onset at one of 13 pediatric MS centers. Controls, less than 20 years old, were recruited from the same centers. Participants’ responses to the validated Block Kids Food Screener questionnaire (NutritionQuest) were used to estimate daily sodium intake. Sodium intake was compared between cases and controls and adjusted for age, race, and insurance status in logistic regression models. 

Results:

Among 122 cases and 202 controls, baseline characteristics were similar for age, mean energy intake (kcal/d), total fat (g/d) and race; however, there were significantly more female and Hispanic/Latino cases compared to controls. Unadjusted dietary sodium intake was not significantly different between cases (1984 mg/d) and controls (2094 mg/d). The mean sodium intake was higher in female cases than controls (1728 mg/d vs. 1677, p=0.89). The percentages of participants exceeding adequate intake of sodium were similar between cases and controls for both males and females. Preliminary analysis adjusting for age, race, and insurance status revealed a trend towards increased odds of MS (OR=1.018) for each 100 mg/d increase in sodium (95% CI 0.994, 1.042, p=0.139). Adjustment of analyses for body mass index is pending. Data from an additional 105 subjects is currently being added to the initial analyses. 

Conclusions:

No significant difference in dietary sodium intake was found between cases and controls in the preliminary analysis. However, the trend toward an increased likelihood of MS with higher salt intake in the adjusted model highlights the need for further investigation of salt as a potential mediator of MS risk in a larger subject pool.